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Exosome

Extracellular exosomes are small extracellular vesicles released by cells into the extracellular space and found in many body fluids, including human plasma, urine, and nipple discharge.

Rebuilt from PubMed 10 Sept 2026 · no new papers today

Where the papers sit

17 papers study exosome directly. Those 17 are one subject: Extracellular Vesicle Biology. Work spans exosome-mediated signaling, tissue repair, disease mechanisms, viral transmission, and biosensing, without a shared mechanistic or clinical direction. No way of splitting those 17 scores better than chance. 1 new direction follows.

NEW DIRECTION

Exosomes function as pathogen-controlled vehicles for infectious viral transmission

Tembusu virus infection in cell culture showed that exosomes are not only host-derived communication, therapeutic, or diagnostic entities: exosomes released by infected cells carried viral genomic RNA and viral proteins into recipient HEK293 cells, suppressed antiviral immune-gene expression, and created a proviral environment supporting productive infection through an NS4A–Rab27a axis. This assigns exosomes a distinct role as virus-directed transmission and immune-evasion vehicles. 42572027Aug

Recent Findings on exosome

Studies report that exosomes and extracellular vesicles from mesenchymal stem cells or adipocyte-like cells modify tissue injury, inflammation, apoptosis, and repair in preclinical models 42663757Aug42638487Aug42480151Jul42436308Jul42302977Jun41997426Apr. Their effects depend on the source, cargo, target cell, and delivery context. Adipocyte-derived exosomes altered lipid metabolism and stemness, while conditioned media produced stronger pro-apoptotic effects than exosomes in MCF-7 cells 42678968Sep. Injury-preconditioned exosomes combined with 3D-CS scaffolds promoted neuroprotection, neurogenesis, angiogenesis, and neurological recovery after traumatic brain injury 42638487Aug. The field is moving toward engineered or preconditioned vesicles, tissue-specific delivery, and defined mechanisms such as ferroptosis, TGF-β-Smad/REDD1 signaling, USP21-dependent autophagy, and immunomodulation.

Studies increasingly pair exosome detection with aptamers, nanocomposites, fluorescent probes, and portable readouts 42611334Aug42520835Jul42215863May. These platforms target the CD63 molecule or disease-associated membrane proteins and report improved selectivity, reproducibility, storage stability, and detection in serum, plasma, or urine 42611334Aug42520835Jul42215863May. Their analytical sensitivity differs substantially, ranging from 3.2 particles/µL for an electrochemical biosensor to 4.58 × 10^6 particles/ml for a smartphone-based assay 42611334Aug42520835Jul. Other studies extend exosome analysis toward clinical sampling and mechanism, including extracellular vesicle miRNAs in nipple discharge and NS4A-Rab27a-dependent Tembusu virus transmission 42307695Jun42572027Aug. The direction is toward rapid, minimally invasive profiling, although the abstracts do not establish common standards for assay performance or clinical validation.

Studies show that exosomal cargo can alter cell migration, proliferation, apoptosis, cell cycle behavior, and macrophage-mediated immune responses 42693330Sep42159846May42162765May42102612May. Exosomal miR-22-3p inhibited p53 in endometrial epithelial cells, whereas menstrual blood stem cell-derived exosomes increased reactive oxygen species, apoptosis, and cell cycle arrest in breast cancer cells 42693330Sep42159846May. In contrast, CD73-expressing exosomes promoted M2 macrophage differentiation and regulatory T cell induction after renal ischemia-reperfusion injury, while M2 macrophage exosomes transferred miR-124 to suppress Calpain-1 and cardiac remodeling 42162765May42102612May. The studies therefore describe both pathogenic and protective effects, depending on vesicle source, recipient cell, and molecular cargo. Aging-related changes in canine extracellular vesicle abundance, size heterogeneity, sterol content, and miRNA levels further support cargo profiling as a direction for biomarker and therapeutic development 42319614Jun.