C. albicans ATCC 10231
Overview
C. albicans ATCC 10231 is a standardized laboratory reference strain of Candida albicans, a medically important opportunistic fungal pathogen. As an ATCC-deposited strain, it is widely used in antifungal susceptibility testing, formulation development, biofilm studies, and host–pathogen interaction experiments. In biomedical research, this strain serves as a reproducible model for evaluating antifungal agents, delivery systems, and anti-virulence strategies against Candida infections.
Biologically, C. albicans is notable for its ability to switch between yeast and hyphal forms, form biofilms, and cause mucosal and invasive disease. In the recent studies provided, C. albicans ATCC 10231 was used as a target organism in assays assessing antifungal activity, including ophthalmic, wound, and polymicrobial infection models. These investigations reflect its role as a benchmark strain for comparing drug potency and formulation performance against C. albicans, often alongside pathogens such as Staphylococcus aureus, Escherichia coli, and Pseudomonas aeruginosa.
Recent Publications Summary
Recent studies involving C. albicans ATCC 10231 focused on antifungal activity, combination therapy, and host-directed approaches. In a marine-fungal natural products study, compounds isolated from Aspergillus sp. WHUF04-170 were tested for antimicrobial effects, and compounds 1 and 15 were reported to enhance the activity of amphotericin B against C. albicans strains, including the standard reference strain ATCC 10231 and drug-resistant clinical isolates 41651325Feb. Another investigation of benzimidazole-based derivatives found that several synthesized compounds had antifungal activity against C. albicans, with compounds 3c and 3i showing particularly strong potency and mechanistic evidence consistent with reactive oxygen species accumulation and ergosterol-related membrane disruption 41831427Mar.
Additional publications examined delivery systems and formulations intended to improve antifungal efficacy against C. albicans. A pH-responsive human serum albumin nanoparticle platform for amphotericin B delivery was reported to provide sustained, acidic pH-dependent release, improved biocompatibility, and retained antifungal activity against C. albicans 42190773May. In a wound-infection context, dissolving microneedles co-loaded with deferoxamine-anchored Ti3C2Tx MXene were shown to exert photothermal antibacterial effects against C. albicans under near-infrared irradiation as part of a broader polymicrobial wound-healing strategy 42119861May. A separate study of melittin in fungal encephalitis found that the peptide directly inhibited C. albicans growth in vitro and attenuated virulence gene expression, while also enhancing blood–brain barrier permeability and reducing fungal burden and neuroinflammation in a murine model 41962469Apr.
Natural product screening also included C. albicans ATCC 10231 in broader antifungal evaluations. A saponin-enriched fraction from Sarcomphalus joazeiro was analyzed by UPLC-QTOF-MS/MS and assessed against Candida spp., including C. albicans, as part of an investigation into plant-derived antifungal potential 42261063Jun. Likewise, a hyaluronate-coated novasome formulation carrying Beta vulgaris subsp. cicla extract was tested against C. albicans in agar well assays, with the crude extract described as having limited antimicrobial activity but some effect against C. albicans 42303711Jun. In another natural-product study, the ethyl acetate extract of Paullinia pinnata and its isolated triterpenoid were evaluated against drug-resistant Candida strains and clinical isolates, including C. albicans, with additional testing of combinations with voriconazole, nystatin, and caspofungin and assessment of antibiofilm activity 42224217Jun.
A vaccine-focused nonhuman primate study also provided context for C. albicans infection biology relevant to recurrent vulvovaginal candidiasis. Japanese macaques were shown to be susceptible to vaginal C. albicans infection, with hyphae formation and polymorphonuclear cell recruitment in the vaginal lumen, and infection or reinfection induced only limited anti-Candida IgG responses that did not prevent reinfection 42371915Jun. Although the abstract does not specify ATCC 10231, it supports the use of C. albicans challenge models to study mucosal infection and vaccine-mediated protection.
What Changes, What Holds
1. Combination and mechanism studies extend the strain’s role as a screening target, not its core identity
REINFORCES These findings keep C. albicans ATCC 10231 in the same established role as a benchmark organism for antifungal testing, while adding more examples of combination-therapy and mechanism-focused screening. The marine natural products work suggests some compounds can potentiate amphotericin B, and the benzimidazole series adds another mechanistic antifungal class with oxidative stress and membrane effects 41651325Feb41831427Mar. That strengthens its utility for comparative drug evaluation without changing what the strain is understood to be.
2. New delivery and host-directed approaches broaden how the strain is used in antifungal models
REINFORCES These studies do not alter the baseline account of C. albicans ATCC 10231 as a reference strain, but they do show it continuing to serve as a test organism in increasingly sophisticated formulation and host-directed settings. The nanoparticle amphotericin B platform and the microneedle/MXene wound strategy both fit the existing pattern of evaluating delivery performance against Candida, while the melittin encephalitis work adds a host-permeability and virulence-suppression angle 42190773May42119861May41962469Apr. The main change is breadth of application, not a new biological role.
3. Plant-derived and formulation-based screens keep confirming the strain’s value in broad antifungal evaluation
REINFORCES These reports reinforce the strain’s established use in natural-product and formulation screening by adding more compounds and extracts to the same comparative testing framework. The saponin fraction, beet-extract novasome, and Paullinia pinnata work all treat C. albicans ATCC 10231 as a standard comparator for antifungal potency and antibiofilm assessment, which is exactly the kind of benchmark use already described in the overview 42261063Jun42303711Jun42224217Jun. Nothing here displaces the baseline; it mainly expands the menu of candidate antifungals.
4. Mucosal infection biology in primates supports the relevance of Candida challenge models, but not strain-specific conclusions
NEW DIRECTION This work adds a host-pathogenesis and vaccine-immunity dimension that the overview does not cover, namely susceptibility to vaginal infection, reinfection, and limited protective IgG responses in a primate model. Because the abstract does not specify ATCC 10231, it cannot be taken as direct evidence about the reference strain itself; instead, it supports the broader use of C. albicans challenge systems for studying recurrent mucosal disease and incomplete immunity 42371915Jun. The baseline stands, but the model context is widened.
Overview update candidates: none.
c. albicans atcc 10231
Background Contexts
In the literature, the biological baseline, pathological conditions, or disease models commonly surrounding c. albicans atcc 10231 are described as follows:
- vulvovaginal candidiasis (Disease) — 3 papers: PMIDs 42371915, 42224217, 41825727
- fungal keratitis (Disease) — 2 papers: PMIDs 42167466, 42150646
- amphotericin B (Therapy) — 1 paper: PMIDs 42190773
- antimicrobial resistance (Other) — 1 paper: PMIDs 42261063
- azole (Therapy) — 1 paper: PMIDs 42371915
- bioactive natural compounds (Other) — 1 paper: PMIDs 42261063
- blood–brain barrier (Biological Process) — 1 paper: PMIDs 41962469
- Chronic diabetic foot ulcers (Disease) — 1 paper: PMIDs 42052713
- Cinnamomum cassia (Organism) — 1 paper: PMIDs 41935478
- Marine-derived fungi (Organism) — 1 paper: PMIDs 41651325
- skin barrier (Other) — 1 paper: PMIDs 42119861
Methodologies & Technologies Used
Researchers utilize the following experimental methods, imaging platforms, computational models, or biological reagents to study c. albicans atcc 10231:
- (RS)-propylene glycol (Chemical) — 1 paper: PMIDs 41903825
- 2,2-diphenyl-1-picrylhydrazyl (Technology) — 1 paper: PMIDs 41935478
- 4-dimethylaminopyridine (Chemical) — 1 paper: PMIDs 42167466
- adolescence (Chemical) — 1 paper: PMIDs 41903825
- Aspergillus sp. WHUF04-170 (Organism) — 1 paper: PMIDs 41651325
- Benzimidazole-2-substituted phenyl alkane sulfonate derivatives (Chemical) — 1 paper: PMIDs 41831427
- C. albicans-induced fungal encephalitis (Disease) — 1 paper: PMIDs 41962469
- Carbopol 934 (Chemical) — 1 paper: PMIDs 42150646
- caspofungin (Therapy) — 1 paper: PMIDs 42224217
- choline chloride:glycerol (Chemical) — 1 paper: PMIDs 41935478
- deep eutectic solvent (Technology) — 1 paper: PMIDs 41935478
- dimethyl sulfoxide (Chemical) — 1 paper: PMIDs 42150646
Molecular Interventions & Targets
The primary molecular pathways, regulatory genes, enzymes, or therapeutic agents actively targeted and manipulated in relation to c. albicans atcc 10231 include:
- Staphylococcus aureus (Organism) — 4 papers: PMIDs 42303711, 42119861, 42019771, 41831427
- amphotericin B (Therapy) — 3 papers: PMIDs 42150646, 41831427, 41651325
- Escherichia coli (Organism) — 3 papers: PMIDs 42303711, 42119861, 42019771
- Candida krusei (Organism) — 2 papers: PMIDs 42261063, 42224217
- Candida tropicalis (Organism) — 2 papers: PMIDs 42261063, 42224217
- (E)-cinnamaldehyde (Chemical) — 1 paper: PMIDs 41935478
- 6α-(3'-methoxy-4'-hydroxybenzoyl)-lup-20(29)-ene-3-one (Chemical) — 1 paper: PMIDs 42224217
- Acinetobacter baumannii (Organism) — 1 paper: PMIDs 41831427
- Amfocare (Therapy) — 1 paper: PMIDs 42150646
- bisabolane-type sesquiterpenes (Chemical) — 1 paper: PMIDs 41651325
- Candida glabrata (Organism) — 1 paper: PMIDs 42224217
- Candida parapsilosis (Organism) — 1 paper: PMIDs 42224217
Observed Outcomes & Phenotypes
The phenotypic changes, physiological endpoints, or clinical metrics observed and measured in connection with c. albicans atcc 10231 include:
- fungal burden (Clinical Metric) — 2 papers: PMIDs 42371915, 41962469
- inflammatory conditions (Biological Process) — 2 papers: PMIDs 42119861, 42052713
- reactive oxygen species (Chemical) — 2 papers: PMIDs 42052713, 41831427
- 2,2'-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) (Other) — 1 paper: PMIDs 41935478
- 2-phenylchromane flavonoid (Chemical) — 1 paper: PMIDs 42261063
- 5 months (Clinical Metric) — 1 paper: PMIDs 41903825
- 50% inhibition concentration (IC50) (Clinical Metric) — 1 paper: PMIDs 42303711
- acid (Chemical) — 1 paper: PMIDs 42261063
- amphotericin B (Therapy) — 1 paper: PMIDs 42224217
- anti-biofilm activity (Biological Process) — 1 paper: PMIDs 42224217
- anti-Candida IgG (Clinical Metric) — 1 paper: PMIDs 42371915
- anti-NXT-2 IgG (Clinical Metric) — 1 paper: PMIDs 42371915
General Takeaways & Clinical Potentials
The high-level concepts, clinical translations, and overarching conclusions proposed in the research surrounding c. albicans atcc 10231 are summarized below:
- antifungal potential (Other) — 1 paper: PMIDs 42261063
- antimicrobial coatings (Other) — 1 paper: PMIDs 42019771
- benzimidazole-based sulfonate hybrids (Chemical) — 1 paper: PMIDs 41831427
- Clinical Recommendations (Other) — 1 paper: PMIDs 42303711
- clinical translation potential (Other) — 1 paper: PMIDs 42167466
- combination regimens (Other) — 1 paper: PMIDs 41651325
- DTM MN treatment (Other) — 1 paper: PMIDs 42119861
- effective strategy for managing complex biofilm-associated infections in diabetic wounds (Other) — 1 paper: PMIDs 42052713
- ergosterol-associated membrane targeting (Biological Process) — 1 paper: PMIDs 41831427
- Fungal infections (Other) — 1 paper: PMIDs 42261063
- healing of infected wounds (Clinical Metric) — 1 paper: PMIDs 42019771
- in situ gelling lipid-based platform (Other) — 1 paper: PMIDs 42150646