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CAV1

CAV1 is the gene associated with caveolin-1, a molecular target investigated across diverse biological and medical contexts.

1 paper landed today · 11 Sept 2026
  • Involvement of phosphorylated caveolin-1 in the dorsal root ganglion in diabetic neuropathic pain and the interventional effect of electroacupuncture. PMID 42714335

Where the papers sit

8 papers study cav1 directly. Those 8 do not group into themes. The group has no shared biological question: it ranges from neuropathic pain and inflammatory bowel disease to cancer drug resistance, genome editing, and computational drug discovery. No common mechanism or research direction emerges. They are no more alike than papers drawn from anywhere in the corpus. 1 new direction follows.

NEW DIRECTION

CAV1 is assigned a developmental ciliary function in an invertebrate model

The Urechis unicinctus CRISPR/Cas9 embryo-editing model 42684483Sep moves CAV1 beyond the set’s disease, treatment-response, and signaling contexts by showing that disruption of the gene produces shortened ciliary structures, altered embryonic expression, and impaired larval swimming. Instead of treating CAV1 primarily as a disease-associated target or response-associated molecular marker, this work places it in cilia formation and organismal development, opening a functional-genetics direction for CAV1 in an emerging aquaculture and evolutionary model.

Recent Findings on CAV1

Cross-Domain Biomedical Research: Phosphorylated CAV1 increased in dorsal root ganglia during diabetic neuropathic pain, while siRNA or electroacupuncture improved thermal hyperalgesia 42714335Sep. CRISPR/Cas9 targeting of Caveolin-1 in Urechis unicinctus disrupted ciliary structure, swimming performance, and embryonic expression patterns 42684483Sep. Multi-omics and network analyses identified CAV1 among markers of ovarian cancer drug resistance and conserved host regulators linking inflammatory pathways, cytoskeletal remodeling, and focal adhesion 42443519Jul42249966Jun. Cancer models generally linked higher CAV1 activity with treatment resistance through CSC glycolysis, PI3K/AKT signaling, or fatty acid β-oxidation 42177885May42087353May41972796Apr. Ulcerative colitis findings instead associated CAV1 upregulation with FAK signaling, epithelial barrier restoration, and mucosal healing, highlighting context-dependent effects 42200545May.